Download e-book for iPad: Adverse Cutaneous Drug Reactions to Cardiovascular Drugs by Esen Özkaya, Kurtuluş Didem Yazganoğlu

By Esen Özkaya, Kurtuluş Didem Yazganoğlu

ISBN-10: 1447165357

ISBN-13: 9781447165354

ISBN-10: 1447165365

ISBN-13: 9781447165361

Adverse cutaneous drug reactions (ACDR) are one of the so much widespread occasions in sufferers receiving drug remedy. Cardiovascular (CV) medications are an immense crew of substances with capability chance of constructing ACDR particularly in aged as advertising of extra new medicines and their prescription proceed to extend. besides the fact that, like with so much different medicinal drugs the precise occurrence of cutaneous unintended effects from CV medicines is tough to estimate because of sporadic reporting. in addition, a competent designation of a undeniable drug because the reason for a undeniable kind of response can hardly be made. except the well known angioedema/urticaria from ACE inhibitors, lichen planus / lichenoid response from beta adrenergic blockers and photosensitivity from thiazid diuretics, ACDR from CV medications may be obvious in a large spectrum extending to infrequent yet life-threatening stipulations comparable to erythroderma, Stevens-Johnson syndrome, poisonous epidermal necrolysis or drug allergy syndrome. during this accomplished evaluate, the suggested sorts of ACDR to CV medicinal drugs may be mentioned based on drug classification and the kind of dermatologic response with exact emphasize on cross-reactions and the function of patch trying out in diagnosis.

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Extra resources for Adverse Cutaneous Drug Reactions to Cardiovascular Drugs

Example text

Some drugs have been reported to exacerbate acne vulgaris. Corticosteroids, epidermal growth factor receptor inhibitors, anticonvulsants, antipsychotics, antidepressants, lithium, TNF-alpha inhibitors, anabolic steroids, danazol, antituberculosis drugs including isoniazid, oral contraceptives, and halogenated drugs are among the main inducers of acneiform drug eruption [35]. Regarding cardiovascular drugs, quinidine may also be associated with acneiform lesions [35]. The condition usually resolves after withdrawal of the causative drug and treatment with appropriate topical and/or systemic drugs such as tetracyclines.

16), and on other intertriginous (axillae) or major flexural (antecubital/popliteal) areas. Lesions are sometimes accompanied by papules, pustules, vesicles, bullae, petechiae, or pustules, the latter raising the question as to whether acute generalized exanthematous pustulosis (AGEP) and BS should not be regarded as variants of a continuous spectrum of possible clinical manifestations [10, 11]. Itching and burning are usually the accompanying symptoms [10]. , mercury and nickel, are the main inducers of BS.

13), and weeping, and in the healing phase with desquamation. The eruption usually starts symmetrically on flexural and intertriginous areas, subsequently evolving into a generalized pattern. In rare cases, it may evolve into erythroderma/exfoliative dermatitis. Histopathologically, spongiosis in the epidermis is the hallmark for acute eczema. Eczematous drug eruption usually occurs with systemic use of contact allergenic drugs or their cross-reactants in patients previously sensitized by topical use of these drugs.

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Adverse Cutaneous Drug Reactions to Cardiovascular Drugs by Esen Özkaya, Kurtuluş Didem Yazganoğlu


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